JAAHA Track the topics, authors and articles important to you
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Mayhew, P. D.
Right arrow Articles by Glass, E. N.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Mayhew, P. D.
Right arrow Articles by Glass, E. N.
Journal of the American Animal Hospital Association 38:262-270 (2002)
© 2002 American Animal Hospital Association

Retrospective Study

Trigeminal Neuropathy in Dogs: A Retrospective Study of 29 Cases (1991–2000)

Philipp D. Mayhew, BVM&S, MRCVS, William W. Bush, VMD and Eric N. Glass, MS, DVM, Diplomate ACVIM (Neurology)

From the Department of Clinical Studies, School of Veterinary Medicine, The University of Pennsylvania, 3900 Delancey Street, Philadelphia, Pennsylvania 19104-6010.

The medical records of 29 dogs unable to close their mouths due to flaccid paralysis or paresis of the muscles innervated by the mandibular branch of the trigeminal nerve, were reviewed. Idiopathic trigeminal neuropathy was diagnosed in 26 dogs based on complete resolution of clinical signs and lack of any long-term neurological disease. Of these dogs, golden retrievers were over-represented. No age, sex, or seasonal predispositions were identified. Trigeminal sensory innervation deficits were observed in 35% (9/26), facial nerve deficits were observed in 8% (2/26), and Horner’s syndrome was observed in 8% (2/26) of dogs. Electromyographic examination of the muscles of mastication revealed abnormalities in seven of nine dogs. Results of cerebrospinal fluid analysis were abnormal in seven of eight dogs. Corticosteroid therapy did not affect the clinical course of the disease. Mean time to recovery was 22 days. Lymphosarcoma, Neospora caninum infection, and severe polyneuritis of unknown origin were diagnosed in three of 29 dogs at necropsy.




This article has been cited by other articles:


Home page
 Journal of the American Animal Hospital AssociationHome page
J. S. Messer, S. O. Wagner, R. D. Baumwart, and C. M. Colitz
A Case of Canine Streptococcal Meningoencephalitis Diagnosed Using Universal Bacterial Polymerase Chain Reaction Assay
J. Am. Anim. Hosp. Assoc., July 1, 2008; 44(4): 205 - 209.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 2002 by the American Animal Hospital Association.